vault backup: 2025-12-14 08:28:15

content/.obsidian/community-plugins.json

@@ -1,14 +1,9 @@
 [
   "obsidian-git",
   "omnisearch",
-  "text-extractor",
   "spoiler-block-obsidian",
-  "pretty-properties",
   "dataview",
   "emoji-shortcodes",
-  "obsidian42-brat",
-  "agent-client",
-  "manual-sorting",
   "obsidian-icon-folder",
-  "image-converter"
+  "obsidian-table-to-csv-exporter"
 ]

content/.obsidian/plugins/obsidian-table-to-csv-exporter/main.js

@@ -0,0 +1,224 @@
+/*
+THIS IS A GENERATED/BUNDLED FILE BY ESBUILD
+if you want to view the source, please visit the github repository of this plugin
+*/
+
+var __create = Object.create;
+var __defProp = Object.defineProperty;
+var __getOwnPropDesc = Object.getOwnPropertyDescriptor;
+var __getOwnPropNames = Object.getOwnPropertyNames;
+var __getProtoOf = Object.getPrototypeOf;
+var __hasOwnProp = Object.prototype.hasOwnProperty;
+var __markAsModule = (target) => __defProp(target, "__esModule", { value: true });
+var __export = (target, all) => {
+  __markAsModule(target);
+  for (var name in all)
+    __defProp(target, name, { get: all[name], enumerable: true });
+};
+var __reExport = (target, module2, desc) => {
+  if (module2 && typeof module2 === "object" || typeof module2 === "function") {
+    for (let key of __getOwnPropNames(module2))
+      if (!__hasOwnProp.call(target, key) && key !== "default")
+        __defProp(target, key, { get: () => module2[key], enumerable: !(desc = __getOwnPropDesc(module2, key)) || desc.enumerable });
+  }
+  return target;
+};
+var __toModule = (module2) => {
+  return __reExport(__markAsModule(__defProp(module2 != null ? __create(__getProtoOf(module2)) : {}, "default", module2 && module2.__esModule && "default" in module2 ? { get: () => module2.default, enumerable: true } : { value: module2, enumerable: true })), module2);
+};
+var __async = (__this, __arguments, generator) => {
+  return new Promise((resolve, reject) => {
+    var fulfilled = (value) => {
+      try {
+        step(generator.next(value));
+      } catch (e) {
+        reject(e);
+      }
+    };
+    var rejected = (value) => {
+      try {
+        step(generator.throw(value));
+      } catch (e) {
+        reject(e);
+      }
+    };
+    var step = (x) => x.done ? resolve(x.value) : Promise.resolve(x.value).then(fulfilled, rejected);
+    step((generator = generator.apply(__this, __arguments)).next());
+  });
+};
+
+// main.ts
+__export(exports, {
+  default: () => Table2CSVPlugin
+});
+var import_obsidian = __toModule(require("obsidian"));
+var DEFAULT_SETTINGS = {
+  exportPath: "./",
+  baseFilename: "table-export",
+  fileNumber: "001",
+  sepChar: "sepChar-semicolon",
+  quoteDataChar: "quoteChar-noQuote",
+  saveToClipboardToo: false,
+  removeCRLF: "removeCRLF-space"
+};
+var Table2CSVPlugin = class extends import_obsidian.Plugin {
+  onload() {
+    return __async(this, null, function* () {
+      yield this.loadSettings();
+      this.addCommand({
+        id: "obsidian-table-to-csv-exporter",
+        name: "Export table to CSV file",
+        checkCallback: (checking) => {
+          const view = this.app.workspace.getActiveViewOfType(import_obsidian.MarkdownView);
+          if (view) {
+            if (!checking) {
+              const viewMode = view.getMode();
+              if (viewMode == "preview") {
+                const csvString = htmlToCSV(view.previewMode.containerEl, this.settings.sepChar, this.settings.quoteDataChar, this.settings.removeCRLF);
+                if (csvString.length > 0) {
+                  const filename = `${this.settings.baseFilename}-${this.settings.fileNumber}.csv`;
+                  this.app.vault.create(filename, csvString).then(() => {
+                    let fn = +this.settings.fileNumber;
+                    fn++;
+                    if (fn == 1e3)
+                      fn = 1;
+                    let newFileNumberString = fn + "";
+                    while (newFileNumberString.length < 3)
+                      newFileNumberString = "0" + newFileNumberString;
+                    this.settings.fileNumber = newFileNumberString;
+                    if (this.settings.saveToClipboardToo) {
+                      navigator.clipboard.writeText(csvString).then(() => {
+                        new import_obsidian.Notice(`The file ${filename} was successfully created in your vault. The contents was also copied to the clipboard.`);
+                      }).catch((err) => {
+                        new import_obsidian.Notice("There was an error with copying the contents to the clipboard.");
+                      });
+                    } else {
+                      new import_obsidian.Notice(`The file ${filename} was successfully created in your vault.`);
+                    }
+                  }).catch((error) => {
+                    const errorMessage = `Error: ${error.message}`;
+                    new import_obsidian.Notice(errorMessage);
+                  });
+                } else {
+                  new import_obsidian.Notice(`No table was found. No CSV file was written.`);
+                }
+              } else {
+                new import_obsidian.Notice("This command only works on panes in reading mode! \u2013 No CSV files were written.");
+              }
+            }
+            return true;
+          }
+          return false;
+        }
+      });
+      this.addSettingTab(new Table2CSVSettingTab(this.app, this));
+      console.log(`Table to CSV plugin: Version ${this.manifest.version} loaded.`);
+    });
+  }
+  onunload() {
+  }
+  loadSettings() {
+    return __async(this, null, function* () {
+      this.settings = Object.assign({}, DEFAULT_SETTINGS, yield this.loadData());
+    });
+  }
+  saveSettings() {
+    return __async(this, null, function* () {
+      yield this.saveData(this.settings);
+    });
+  }
+};
+function htmlToCSV(html, sepMode, quoteChar, removeCRLF) {
+  var data = [];
+  var table = html.querySelector("table");
+  if (table) {
+    var rows = table.rows;
+    for (var i = 0; i < rows.length; i++) {
+      var row = [], cols = rows[i].querySelectorAll("td, th");
+      for (var j = 0; j < cols.length; j++) {
+        var cellContent = cols[j].innerText;
+        if (removeCRLF == "removeCRLF-clear") {
+          cellContent = cellContent.replace(/(\r\n|\n|\r)/gm, "");
+        } else if (removeCRLF == "removeCRLF-space") {
+          cellContent = cellContent.replace(/(\r\n|\n|\r)/gm, " ");
+        } else if (removeCRLF == "removeCRLF-string1") {
+          cellContent = cellContent.replace(/(\r\n|\n|\r)/gm, "[CR]");
+        }
+        if (quoteChar == "quoteChar-doubleQuotes") {
+          cellContent = '"' + cellContent + '"';
+        } else if (quoteChar == "quoteChar-singleQuotes") {
+          cellContent = "'" + cellContent + "'";
+        }
+        row.push(cellContent);
+      }
+      var sepChar = ";";
+      switch (sepMode) {
+        case "sepChar-semicolon":
+          sepChar = ";";
+          break;
+        case "sepChar-comma":
+          sepChar = ",";
+          break;
+        case "sepChar-tab":
+          sepChar = "	";
+          break;
+        case "sepChar-pipe":
+          sepChar = "|";
+          break;
+        case "sepChar-tilde":
+          sepChar = "~";
+          break;
+        case "sepChar-caret":
+          sepChar = "^";
+          break;
+        case "sepChar-colon":
+          sepChar = ":";
+          break;
+      }
+      data.push(row.join(sepChar));
+    }
+  }
+  if (data.length > 0)
+    return data.join("\n");
+  else
+    return "";
+}
+var Table2CSVSettingTab = class extends import_obsidian.PluginSettingTab {
+  constructor(app, plugin) {
+    super(app, plugin);
+    this.plugin = plugin;
+  }
+  display() {
+    const { containerEl } = this;
+    containerEl.empty();
+    containerEl.createEl("h2", { text: "Settings for the Table to CSV Plugin." });
+    containerEl.createEl("p", { text: "NOTE: Currently, the exported CSV files are saved inside your vault main folder." });
+    new import_obsidian.Setting(containerEl).setName("CSV file base filename").setDesc('Enter the base filename. The "File Number addendum" gets added after that and finally .csv').addText((text) => text.setPlaceholder("<enter a base filename").setValue(this.plugin.settings.baseFilename).onChange((value) => __async(this, null, function* () {
+      this.plugin.settings.baseFilename = value;
+      yield this.plugin.saveSettings();
+    })));
+    new import_obsidian.Setting(containerEl).setName("File Number addendum").setDesc("This number gets added to the base filename and incremented after each export. Normally, you shouldn't need to edit this.").addText((text) => text.setPlaceholder("").setValue(this.plugin.settings.fileNumber).onChange((value) => __async(this, null, function* () {
+      this.plugin.settings.fileNumber = value;
+      yield this.plugin.saveSettings();
+    })));
+    new import_obsidian.Setting(containerEl).setName("Data fields separation character/string").setDesc("This character will be put between each cell's data. Defaults to a semicolon.").addDropdown((dropdown) => dropdown.addOption("sepChar-semicolon", "; (semicolon)").addOption("sepChar-comma", ", (comma)").addOption("sepChar-tab", "\\t (tab)").addOption("sepChar-pipe", "| (pipe)").addOption("sepChar-tilde", "~ (tilde)").addOption("sepChar-caret", "^ (caret)").addOption("sepChar-colon", ": (colon)").setValue(this.plugin.settings.sepChar).onChange((value) => __async(this, null, function* () {
+      this.plugin.settings.sepChar = value;
+      yield this.plugin.saveSettings();
+    })));
+    new import_obsidian.Setting(containerEl).setName("Quote data").setDesc("Do you want quotation marks around each cell's data?").addDropdown((dropdown) => dropdown.addOption("quoteChar-noQuote", "Don't quote data").addOption("quoteChar-doubleQuotes", 'Quote data with double quote character (")').addOption("quoteChar-singleQuotes", "Quote data with single quote character (')").setValue(this.plugin.settings.quoteDataChar).onChange((value) => __async(this, null, function* () {
+      this.plugin.settings.quoteDataChar = value;
+      yield this.plugin.saveSettings();
+    })));
+    new import_obsidian.Setting(containerEl).setName("Handling of CR/LF in data").setDesc("Choose how to handle the occurance of return and linefeed characters in data cells.").addDropdown((dropdown) => dropdown.addOption("removeCRLF-clear", "Remove all CR & LF characters").addOption("removeCRLF-space", "Replace all CR & LF characters with one space").addOption("removeCRLF-string1", "Replace all CR & LF characters with string [CR]").setValue(this.plugin.settings.removeCRLF).onChange((value) => __async(this, null, function* () {
+      this.plugin.settings.removeCRLF = value;
+      yield this.plugin.saveSettings();
+    })));
+    new import_obsidian.Setting(containerEl).setName("Copy to clipboard, too").setDesc("Do you want to copy the contents of the CSV file to the system clipboard, too?").addToggle((toggle) => toggle.setValue(this.plugin.settings.saveToClipboardToo).onChange((value) => __async(this, null, function* () {
+      this.plugin.settings.saveToClipboardToo = value;
+      yield this.plugin.saveSettings();
+    })));
+  }
+};
+
+
+/* nosourcemap */

content/.obsidian/plugins/obsidian-table-to-csv-exporter/manifest.json

@@ -0,0 +1,10 @@
+{
+	"id": "obsidian-table-to-csv-exporter",
+	"name": "Table to CSV Exporter",
+	"version": "0.1.4",
+	"minAppVersion": "0.14.6",
+	"description": "This plugin allows for exporting tables from a pane in reading mode into CSV files.",
+	"author": "Stefan Wolfrum",
+	"authorUrl": "https://twitter.com/metawops",
+	"isDesktopOnly": false
+}

content/.obsidian/workspace.json

@@ -55,12 +55,12 @@
             "state": {
               "type": "markdown",
               "state": {
-                "file": "Biokemi/Behöver göra.md",
-                "mode": "source",
+                "file": "Biokemi/Regleringar.md",
+                "mode": "preview",
                 "source": false
               },
               "icon": "lucide-file",
-              "title": "Behöver göra"
+              "title": "Regleringar"
             }
           }
         ],
@@ -121,7 +121,8 @@
       }
     ],
     "direction": "horizontal",
-    "width": 300
+    "width": 300,
+    "collapsed": true
   },
   "right": {
     "id": "2d28508435d0e696",
@@ -223,7 +224,8 @@
       }
     ],
     "direction": "horizontal",
-    "width": 394.5
+    "width": 200,
+    "collapsed": true
   },
   "left-ribbon": {
     "hiddenItems": {
@@ -240,6 +242,11 @@
   },
   "active": "0ee86d2943a05d44",
   "lastOpenFiles": [
+    "table-export-001.xlsx",
+    "lu32604h9hpy3.tmp",
+    "table-export-001.csv",
+    "Biokemi/Behöver göra.md",
+    "Biokemi/Regleringar.md",
     "Biokemi/Metabolism/Biokemi ur ett evolutionsperspektiv/Slides.pdf.pdf",
     "has.txt",
     "all.txt",
@@ -249,10 +256,7 @@
     "TaskNotes/Views/mini-calendar-default.base",
     "TaskNotes/Views/kanban-default.base",
     "TaskNotes/Views/calendar-default.base",
-    "TaskNotes/Views/agenda-default.base",
     "TaskNotes/Tasks/ETK-kortlek imorgon kl 09.md",
-    "TaskNotes/Views",
-    "TaskNotes/Tasks",
     "TYK/Presentation.md",
     "Biokemi/Plasmidlabb/Provfrågor.md",
     "Biokemi/Metabolismseminarie/Direktiv.md",
@@ -275,8 +279,6 @@
     "Biokemi/Metabolism/Elektrontransportkedjan/📚 Instuderingsuppgifter.md",
     "Biokemi/Metabolism/Elektrontransportkedjan/🎯 Lärandemål.md",
     "Biokemi/Metabolism/Elektrontransportkedjan/👨🏻‍🏫 Slides.md",
-    "Biokemi/Metabolism/Elektrontransportkedjan/❓ Provfrågor.md",
-    "Biokemi/Metabolism/Diagram/Vägar.md",
     "Biokemi/Metabolism/Diagram/TCA_cycle.svg",
     "Biokemi/Metabolism/Diagram/Glycolysis_overview.svg",
     "Biokemi/Plasmidlabb/Bilder/IMG_1976.jpg",

content/.~lock.table-export-001.csv#

@@ -0,0 +1 @@
+,johandahlin,Johans-M1-Laptop.local,14.12.2025 08:23,file:///Users/johandahlin/Library/Application%20Support/LibreOffice/4;

content/table-export-001.csv

@@ -0,0 +1,71 @@
+Föreläsning;Typ av reglering;Enzym / Protein;Substrat / Hormon / Faktor;Organ;Organell;Anabol / Katabol / När / Varför
+Intro Metabolism / Citratcykeln;Allosterisk (Feedback);Pyruvatdehydrogenas (PDH);Acetyl-CoA (−);Alla mitokondriella;Mitokondrie (Matrix);Katabol. Produkten ansamlas → bromsar inflöde från pyruvat (t.ex. vid β-oxidation).
+Intro Metabolism / Citratcykeln;Allosterisk (Feedback);Pyruvatdehydrogenas (PDH);NADH (−);Alla mitokondriella;Mitokondrie (Matrix);Katabol. Hög reduktionsgrad/ETC “mättad” → bromsar PDH.
+Intro Metabolism / Citratcykeln;Allosterisk (Energikvot);Pyruvatdehydrogenas (PDH);ATP (−);Alla mitokondriella;Mitokondrie (Matrix);Katabol. Hög energi → mindre behov av acetyl-CoA in i CTK.
+Intro Metabolism / Citratcykeln;Allosterisk (Energikvot);Pyruvatdehydrogenas (PDH);ADP (+);Alla mitokondriella;Mitokondrie (Matrix);Katabol. Energifattigt → ökar flödet från pyruvat in i CTK.
+Intro Metabolism / Citratcykeln;Kovalent (Fosforylering);Pyruvatdehydrogenas (PDH);PDH-Kinas (PDK);Alla mitokondriella;Mitokondrie (Matrix);PDK fosforylerar PDH → PDH inaktivt (bromsar glukosoxidation).
+Intro Metabolism / Citratcykeln;Kovalent (Defosforylering);Pyruvatdehydrogenas (PDH);PDH-Fosfatas (PDP);Alla mitokondriella;Mitokondrie (Matrix);PDP defosforylerar PDH → PDH aktivt (ökar pyruvat→acetyl-CoA).
+Intro Metabolism / Citratcykeln;Allosterisk (påverkar PDK);PDH-Kinas (PDK);ATP (+);Alla mitokondriella;Mitokondrie (Matrix);Hög energi → PDK aktivt → PDH stängs (sparar glukos).
+Intro Metabolism / Citratcykeln;Allosterisk (påverkar PDK);PDH-Kinas (PDK);Acetyl-CoA & NADH (+);Alla mitokondriella;Mitokondrie (Matrix);Produkter aktiverar PDK → förstärker PDH-avstängning (feedback-loop).
+Intro Metabolism / Citratcykeln;Allosterisk (påverkar PDK);PDH-Kinas (PDK);Pyruvat (−);Alla mitokondriella;Mitokondrie (Matrix);Högt pyruvat hämmar PDK → håller PDH aktivt (feedforward).
+Intro Metabolism / Citratcykeln;Allosterisk (påverkar PDP);PDH-Fosfatas (PDP);Ca²⁺ (+);Muskler m.fl.;Mitokondrie (Matrix);Muskelarbete → Ca²⁺ → PDP på → PDH på → mer ATP.
+Intro Metabolism / Citratcykeln;Hormonell (påverkar PDP);PDH-Fosfatas (PDP);Insulin (+);Lever / Fettväv;Mitokondrie (Matrix);Fed state: PDH på → acetyl-CoA till CTK/lipogenes.
+Glykolysen;Allosterisk (Feedback);Hexokinas;Glukos-6-fosfat (−);De flesta vävnader (ej leverns glukokinas);Cytosol;Katabol. Förhindrar “inlåsning” av glukos när nedströms är fullt.
+Glykolysen;Substrat/isoenzym;Glukokinas;Glukos (kräver hög [glukos]);Lever (± β-cell);Cytosol;Katabol/anabol koppling. “Tar hand om” glukos efter måltid.
+Glykolysen;Allosterisk (Energikvot);PFK-1;ATP (−);Alla (främst muskel/lever);Cytosol;Katabol. Hög energi → bromsar hastighetsbestämmande steg.
+Glykolysen;Allosterisk (Energikvot);PFK-1;AMP (+);Alla (främst muskel);Cytosol;Katabol. Låg energi → stark aktivering av glykolys.
+Glykolysen;Allosterisk (koppling CTK);PFK-1;Citrat (−);Lever m.fl.;Cytosol;Katabol. CTK “full” → minska inflöde från glykolys.
+Glykolysen;Allosterisk (pH);PFK-1;H⁺ / lågt pH (−);Muskel;Cytosol;Katabol. Skydd mot kraftig acidos vid anaerobt arbete.
+Glykolysen;Allosterisk (potent);PFK-1;Fruktos-2,6-bisfosfat (+);Lever;Cytosol;Katabol. Överstyr ATP-hämning vid högt blodsocker.
+Glykolysen;Allosterisk (Feedforward);Pyruvatkinas;Fruktos-1,6-bisfosfat (+);Lever/muskel;Cytosol;Katabol. Synkar sista steget med högt flöde genom PFK-1.
+Glykolysen;Allosterisk;Pyruvatkinas;ATP (−) / Alanin (−, lever);Lever/muskel;Cytosol;Katabol. Hög energi/byggstenar → bromsar pyruvatproduktion.
+Glykolysen;Hormonell (Kovalent);Pyruvatkinas (L-form);Glukagon → PKA → fosforylering (−);Lever;Cytosol;Fasta: stänger glykolys i lever → sparar substrat för glukoneogenes.
+Glukoneogenes;Allosterisk;Pyruvatkarboxylas;Acetyl-CoA (+);Lever/Njure;Mitokondrie;Anabol. Fettförbränning ger acetyl-CoA → “energi finns” för GNG.
+Glukoneogenes;Allosterisk (Energikvot);Pyruvatkarboxylas;ADP (−);Lever/Njure;Mitokondrie;Anabol. Energibrist → stoppar ATP-krävande glukosnybildning.
+Glukoneogenes;Allosterisk;Fruktos-1,6-bisfosfatas (FBPase-1);Fruktos-2,6-bisfosfat (−);Lever;Cytosol;Anabol. Fed state hämmar GNG via F2,6BP.
+Glukoneogenes;Allosterisk;Fruktos-1,6-bisfosfatas (FBPase-1);AMP (−);Lever;Cytosol;Anabol. Låg energi → stänger GNG.
+Glukoneogenes;Allosterisk;Fruktos-1,6-bisfosfatas (FBPase-1);Citrat (+);Lever;Cytosol;Anabol. Energiöverskott → gynnar glukosnybildning.
+Glukoneogenes / Glykolys;Hormonell (bifunktionellt enzym);PFK-2 / FBPase-2;Insulin → defosforylering → PFK-2 aktiv (↑F2,6BP);Lever;Cytosol;Fed state: ↑ glykolys, ↓ glukoneogenes.
+Glukoneogenes / Glykolys;Hormonell (bifunktionellt enzym);PFK-2 / FBPase-2;Glukagon → PKA → fosforylering → FBPase-2 aktiv (↓F2,6BP);Lever;Cytosol;Fasta: ↓ glykolys, ↑ glukoneogenes.
+Citronsyracykeln;Allosterisk;Citratsyntas;ATP, NADH, Citrat, Succinyl-CoA (−);Alla;Mitokondrie (Matrix);Katabol. Hög energi/produktansamling → bromsar inflöde.
+Citronsyracykeln;Allosterisk;Citratsyntas;ADP (+);Alla;Mitokondrie (Matrix);Katabol. Energifattigt → ökar CTK-flöde.
+Citronsyracykeln;Allosterisk (rate-limiting);Isocitratdehydrogenas;ATP, NADH (−);Alla;Mitokondrie (Matrix);Katabol. Energirikt → bromsar hastighetsbestämmande steg.
+Citronsyracykeln;Allosterisk;Isocitratdehydrogenas;ADP, Ca²⁺ (+);Alla (Ca²⁺ särskilt muskel);Mitokondrie (Matrix);Katabol. Muskelarbete/ADP → mer NADH till ETC.
+Citronsyracykeln;Produktinhibition;α-ketoglutaratdehydrogenas;Succinyl-CoA, NADH (−);Alla;Mitokondrie (Matrix);Katabol. Produkter bromsar komplexet.
+Citronsyracykeln;Allosterisk;α-ketoglutaratdehydrogenas;Ca²⁺ (+);Alla;Mitokondrie (Matrix);Katabol. Ca²⁺ vid arbete → ökar flöde.
+Glykogen;Allosterisk (isoenzym);Glykogenfosforylas (muskel);AMP (+);Muskel;Cytosol;Katabol. Akut energibrist → glykogenolys utan hormonkrav.
+Glykogen;Allosterisk (isoenzym);Glykogenfosforylas (lever);Glukos (−);Lever;Cytosol;Katabol. Högt glukos → stänger glykogenolys i lever.
+Glykogen;Hormonell (kaskad);Glykogenfosforylas;Glukagon/Adrenalin → cAMP → PKA → kinas;Lever/Muskel;Cytosol;Katabol. Fosforylas a (fosforylerad) = aktiv → mobiliserar glukos.
+Glykogen;Hormonell;Glykogenfosforylas;Insulin → PP1 (defosforylering);Lever/Muskel;Cytosol;Fed state: fosforylas b = mindre aktivt → stoppar nedbrytning.
+Glykogen;Hormonell;Glykogensyntas;Insulin → PP1 + GSK3-hämning;Lever/Muskel;Cytosol;Anabol. Defosforylerat syntas (a) = aktivt → lagrar glukos.
+Glykogen;Hormonell;Glykogensyntas;Glukagon/Adrenalin → PKA/GSK3 (fosforylering);Lever/Muskel;Cytosol;Fasta/stress: syntas fosforyleras → inaktivt (hindrar lagring).
+Glykogen;Allosterisk;Glykogensyntas (b-form);Glukos-6-fosfat (+);Lever/Muskel;Cytosol;Anabol. Hög G6P kan driva lagring trots fosforylering.
+Glykogen;Organ-specifik;Glukos-6-fosfatas;Finns i lever (inte muskel);Lever;ER;Varför: lever kan höja blodglukos; muskel använder lokalt.
+Fettsyrasyntes;Allosterisk;Acetyl-CoA-karboxylas (ACC);Citrat (+);Lever/Fettväv;Cytosol;Anabol. Citrat signalerar överskott → ACC aktiveras (polymerisering).
+Fettsyrasyntes;Allosterisk (feedback);Acetyl-CoA-karboxylas (ACC);Palmitoyl-CoA (−);Lever/Fettväv;Cytosol;Anabol. Slutprodukt bromsar → undviker överproduktion.
+Fettsyrasyntes;Hormonell (kovalent);ACC;Insulin (+ via fosfatas);Lever/Fettväv;Cytosol;Anabol. Defosforylering → ACC aktivt → malonyl-CoA ↑.
+Fettsyrasyntes;Energisensor (kovalent);ACC;AMPK (− via fosforylering); AMP (+ på AMPK);Lever/Fettväv;Cytosol;Anabol av. Låg energi → stoppar lipogenes, sparar ATP.
+Fettsyrasyntes;Hormonell (via AMPK);ACC;Glukagon/Adrenalin (−);Lever/Fettväv;Cytosol;Fasta/stress: minskar lipogenes, gynnar mobilisering/oxidation.
+Betaoxidation / Lipolys;Hormonell (kovalent);Hormonkänsligt lipas (HSL);Adrenalin/Glukagon → PKA (+);Fettväv;Cytosol;Katabol. Fosforylering → lipolys ↑ (frisätter FFA).
+Betaoxidation / Lipolys;Hormonell (tillgänglighet);Perilipin;PKA (+);Fettväv;Lipiddroppe;Katabol. Gör TAG mer åtkomligt för lipaser.
+Betaoxidation / Lipolys;Hormonell;HSL (indirekt);Insulin (−; ↓cAMP, ↑fosfatas);Fettväv;Cytosol;Fed state: bromsar lipolys → lagring.
+Betaoxidation;Allosterisk (transport);Karnitin-acyltransferas 1 (CPT1/CAT1);Malonyl-CoA (−);Lever/muskel;Yttre mitokondriemembran;Katabol. Hindrar import när lipogenes pågår (undviker futile cycle).
+Kolesterolsyntes;Transkriptionell;HMG-CoA-reduktas (HMGR);Lågt kolesterol (+ via SREBP);Lever;ER→Golgi→Kärna;Anabol. Lågt kolesterol → SREBP aktiveras → ↑ enzymesyntes/LDL-upptag.
+Kolesterolsyntes;Transkriptionell (feedback);SREBP/SCAP/Insig;Högt kolesterol (−);Lever;ER;Anabol bromsas. SREBP hålls kvar i ER → mindre genuttryck.
+Kolesterolsyntes;Proteolys;HMGR;Steroler/intermediärer (−; ubiquitin/proteasom);Lever;ER-membran;Anabol bromsas snabbt genom nedbrytning av HMGR.
+Kolesterolsyntes;Energisensor (kovalent);HMGR;AMPK fosforylering (−);Lever;ER;Låg energi → stoppar kolesterolsyntes (ATP/NADPH-spar).
+Kolesterolsyntes;Hormonell (kovalent);HMGR;Insulin (+ via defosforylering);Lever;ER;Fed state: syntes på.
+Kolesterolsyntes;Hormonell (via AMPK);HMGR;Glukagon (−);Lever;ER;Fasta: syntes av.
+Ureacykeln;Allosterisk (obligatorisk);Karbamoylfosfatsyntetas 1 (CPS1);N-acetylglutamat (+);Lever;Mitokondrie (Matrix);Katabol/anabol koppling. Avgiftar kväve; utan NAG fungerar CPS1 i praktiken inte.
+Ureacykeln;Substrat-aktivering;N-acetylglutamatsyntas (NAGS);Arginin (+);Lever;Mitokondrie;När protein/AA-nedbrytning hög → arginin ↑ → NAG ↑ → ureacykel upp.
+Pentosfosfatvägen;Allosterisk (feedback);G6PD;NADPH (−);Lever, erytrocyter;Cytosol;Anabol. Om NADPH redan är högt → bromsar PPP.
+Pentosfosfatvägen;Allosterisk (substratstyrd);G6PD;NADP⁺ (+);Lever, erytrocyter;Cytosol;Anabol. NADP⁺ driver PPP när NADPH behövs (t.ex. ROS-skydd i RBC).
+Elektrontransportkedjan/OXPHOS;“Respiratory control”;ATP-syntas / ETC (kopplat);ADP (+);Alla aeroba vävnader;Inre mitokondriemembran;Katabol. ADP-tillgång styr OXPHOS-hastighet (”acceptor control”).
+Elektrontransportkedjan/OXPHOS;Substrat/acceptor;Cyt c-oxidas (Komplex IV);O₂ (+ som slutacceptor);Alla aeroba;Inre mitokondriemembran;Katabol. Utan O₂ stannar ETC → NADH↑ → TCA/PDH bromsas.
+Ketonkroppar;Substratflöde (indirekt);Ketogenes (väg);OAA “dras” till GNG; acetyl-CoA ansamlas;Lever;Mitokondrie (Matrix);Katabol. Fasta/insulinbrist: acetyl-CoA → ketoner för perifera vävnader.
+Hemoglobin;Allosterisk (kooperativitet);Hemoglobin;O₂ (+);Erytrocyter;Cytosol;Varför: sigmoidal kurva → effektivt upptag i lungor, avlämning i vävnad.
+Hemoglobin;Allosterisk (Bohr-effekt);Hemoglobin;H⁺/CO₂ (− på O₂-affinitet);Erytrocyter;Cytosol;Varför: surt/CO₂-rikt i vävnad → stabiliserar T-state → O₂ släpps.
+Hemoglobin;Allosterisk;Hemoglobin;2,3-BPG (− på O₂-affinitet);Erytrocyter;Cytosol;Varför: underlättar O₂-avlämning; ökar vid höjd/anemi.
+DNA-replikation;Cellcykelreglering;ORC/Cdc6/Cdt1 (licensing);Lågt CDK (G1);Alla delande celler;Kromatin (kärna);Anabol. Tillåter laddning av MCM (förhindrar re-replikation).
+DNA-replikation;Cellcykelreglering;CMG-helikas / DNA-pol;Högt CDK (S);Alla delande celler;Kromatin (kärna);Anabol. Startar replikation och blockerar ny licensing.
+Hemesyntes;Translationell;ALA-syntas;Järnstatus (Fe);Erytroid vävnad;Cytosol/mito-axel;Anabol. Vid järnbrist hämmas effektivt hemsyntes (undviker porfyrin-ack).